Hairy Cell Leukaemia (HCL) (BSH 2020)
BRAF V600E Mutation
Immunohist: CD72+, TRAP+
Flow: CD19+, CD20+, CD22+, CD11c+, CD25+, CD103+, CD123+, Annexin A1+, Cyclin D1+
CD5-, CD23-, CD27-, CD38-
Intro
Uncommon – 6-8x rarer than CLL
2% of all leukaemias, 8% of all mature B/T cell LPDs
Male > Female 4.5:1
Median Age at diagnosis: 58 years
Clinical Presentation
Incidental – FBC performed for other reasons
Symptoms of cytopenias – usually recurrent infection
Typically 1-2 cytopenias present, monocytopenia is almost always present
Splenomegaly is common
differential diagnosis
Hairy Cell Leukaemia Variant, Splenic Marginal Zone Lymphoma, B-PLL, Splenic diffuse red pulp small B cell lymphoma (SDRPL)
Diagnostic Tests
Film
Hairy cells. Twice the size of normal lymphocytes with round, oval or kidney bean shaped nucleus. Loose chromatin. Cytoplasmic projections (hairy). Monocytopenia.
BM Biopsy
90% dry tap, but trephine often fibrotic & hypercellular with many ‘fried egg’ cells. ‘Blood lake’ appearance describes pseudo-sinus formation with extravasation of red cells into the affected areas.
Immunohistochemistry: CD20, DBA44, Annexin A1, CD25, Cyclin D1, SOX11, TRAP, BRAFV600E protein.
Cyclin D1+ in 50%, CD10 in 20%
Flow
‘Hairy Cell Panel’ = CD11c, CD25, CD103, CD123 —> 3 out of 4 distinguishes HCL from other B-LPD.-
Also: CD20+, CD22+, TBX21+, Annexin A1+, FMC7+, CD200+, Cyclin D1+, CD27-, CD38-
Annexin A1 is specific, not expressed in other B cell lymphomas
Post treatment FSc/SSc plot will look unchanged because with the hairy cells gone, normal monocytes will re-populate and fill the same space on the graph.
Molecular
BRAF V600E mutation is a disese-defining event, present in virtually all cases.
Usually IGHV mutated
MRD Monitoring – best done by flow, but test only as good as the sample provided (dry taps)
Staging
No agreed system of staging
CT staging not essential. Though the 10% pts with abdominal LN have poorer response
PR rather than CR after purine analogue treatment is a very poor prognostic sign
Prognosis
Median PFS 16 years
OS is close to that of age-matched controls (compared to median OS of 4 years in 1974)
Death from HCL itself very rare
Treatment
W&W reasonable if asymptomatic with minimal cytopenias
Consider asymptomatic patients for treatment if significant neutropenia + monocytopenia
Direct patients to support services (e.g. Cancer Research UK)
1st Line: Purine Analogues
Irradiated blood products
Pentostatin and Cladribine both have >80% CR rate with >10 year disease free survival
Pentostatin – every fortnight until maximum response. Need normal renal function
Cladribine – many routes/regimens. SC daily for 5 days is the simplest. Rash common
Aciclovir + Septrin prophylaxis
Assess disease response
Rpt BMAT 4-6 months after cladribine
CR = absence of hairy cells from blood and bone marrow + normal FBC + no organomeg.
PR = normal FBC + 50% improvement in organomegaly or bone marrow infiltration
If PR, re-treat with cladribine +/- Rituximab
Treatment of Relapsed/Refractory Disease (Approx 50% of patients)
Offer clinical trial
CR lasting >2 years:
Re-treat with same agent & can add Rituximab to either
CR lasting <2 years:
Whichever purine analogue wasn’t use first line
Moxetumumab pasudotox (Anti-CD22 + pseudomonas exotoxin)
NICE decision expected late in 2020
SE: HUS, Capillary leak syndrome
BRAF inhibitors (Vemurafenib, Dabrafenib)
High efficacy, access via trial/compassionate access
R-Bendamustine
Ibrutinib
Other treatments
Interferon alpha – might use if rapid count recovery required, e.g. presenting with severe infection.
Splenectomy – Rarely required. May be indicated if >10cm below costal margin + low-level BM infiltration
Management in Pregnancy (from previous 2011 guideline)
Prevalence extremely low given demographics of HCL
Avoid treatment if asymptomatic
Interferon alpha if treatment is unavoidable
Hairy Cell Leukaemia-variant (HCL-v)
Unrelated to HCL
Rare
Does not respond to interferon alpha
Poorer responses to cladribine and pentostatin
Different to HCL in that:
Leukocytosis, WBC usually 40-60
No monocytopenia
Cells are villous and large with nucleolus resembling B-PLL
CD11c+, CD25-, CD103-, HC2-
MAP2K1 mutation is the most common molecular finding (BRAFV600E mutation not present)
No adequate treatment.
Cladribine + Rituximab recommended first line.
Moxetumumab pasudotox & Ibrutinib both shown to have activity. Not currently routinely available.
Splenectomy may have a palliative role.